- Excess cortisol production.
- Autoimmune destruction of the adrenal cortex, leading to deficiency of cortisol and aldosterone.
- Excess aldosterone production.
- Impaired glucose metabolism.
No category found.
- Oral antihistamine.
- Immediate administration of epinephrine.
- Topical steroid cream.
- Observation.
- Decreased oncotic pressure.
- Increased hydrostatic pressure in the pulmonary capillaries due to left ventricular dysfunction.
- Increased lymphatic drainage.
- Decreased capillary permeability.
- Degeneration of articular cartilage.
- Autoimmune inflammation of the synovial membrane.
- Deposition of uric acid crystals.
- Bacterial infection of the joint.
- Peptic ulcer.
- Pancreatitis.
- Gallstone obstruction of the cystic duct (biliary colic).
- Appendicitis.
- Hypovolemia.
- Bilateral ureteral obstruction.
- Acute tubular necrosis (ATN) due to prolonged renal ischemia.
- Glomerulonephritis.
- Administering high-flow oxygen.
- Initiating non-invasive positive pressure ventilation (NIPPV) or mechanical ventilation.
- Encouraging deep breathing exercises.
- Prescribing oral antibiotics.
- Direct inflammation of the renal parenchyma.
- Distension of the renal capsule and ureteral spasm.
- Ischemia of the kidney.
- Nerve compression in the spinal cord.
- Abnormal chloride channel function, leading to thick, viscous secretions.
- Impaired ciliary movement.
- Alveolar destruction.
- Bronchial hypersensitivity.
- Increases reabsorption of sodium and water.
- Blocks aldosterone receptors.
- Inhibits sodium and chloride reabsorption in the loop of Henle, leading to increased diuresis.
- Decreases heart rate.
- Glomerular hyperfiltration.
- Direct tubular damage from myoglobin casts.
- Increased erythropoietin production.
- Renal artery stenosis.
- Complement activation.
- Autoantibody production.
- Massive release of histamine and other inflammatory mediators from mast cells and basophils.
- T-cell mediated cytotoxicity.
- Oral analgesics.
- Needle decompression for tension pneumothorax.
- Inhaled bronchodilator.
- IV antibiotics.
- Acute inflammation of nerve endings.
- Demyelination due to autoimmune attack.
- Microvascular damage and metabolic alterations affecting nerve fibers.
- Compression of spinal nerves.
- Loss of dopaminergic neurons.
- Accumulation of amyloid plaques and neurofibrillary tangles.
- Demyelination of nerve fibers.
- Increased cerebrospinal fluid pressure.
- Peptic ulcer.
- Gastric cancer.
- Barrett's esophagus (metaplasia).
- Esophageal varices.
- Increased metabolic rate.
- Decreased production of thyroid hormones, leading to a generalized hypometabolic state.
- Autoimmune destruction of the adrenal glands.
- Excess growth hormone.
- Acute myocardial infarction.
- Esophageal rupture.
- Aortic dissection, requiring urgent surgical intervention.
- Pulmonary embolism.
- T-cell mediated destruction of tissues.
- Autoantibody production and immune complex deposition.
- IgE-mediated hypersensitivity reaction.
- Non-inflammatory joint degeneration.
