- Encouraging vigorous exercise.
- Implementing strict infection control measures and monitoring for signs of infection.
- Offering high-fiber foods.
- Limiting fluid intake.
No category found.
- Respiratory alkalosis.
- The body's attempt to compensate for metabolic acidosis by blowing off CO2?.
- Hypoglycemia.
- Metabolic alkalosis.
- Increased appetite.
- New onset of cyanosis or increased respiratory distress.
- Increased activity level.
- Mild nasal congestion.
- Administering oral corticosteroids.
- Immediately administering a short-acting beta-agonist (SABA) via nebulizer or metered-dose inhaler with spacer.
- Encouraging the child to lie down.
- Taking a detailed history of the child's allergies.
- Oral rehydration solution (ORS) administration.
- Intravenous (IV) fluid bolus of isotonic solution (e.g., normal saline).
- Administration of an antiemetic.
- Administration of an antipyretic.
- To prevent infection at the injection site.
- To ensure proper absorption and prevent lipohypertrophy.
- To make the injections less painful.
- To reduce the cost of insulin.
- Administering antibiotics immediately.
- Providing humidified oxygen, ensuring adequate hydration, and frequent suctioning to clear secretions.
- Placing the infant in Trendelenburg position.
- Encouraging vigorous play.
- Assessing the child's level of consciousness and responsiveness.
- Asking the parents about the last time the child ate.
- Determining the exact onset and progression of symptoms, and assessing respiratory effort for signs of impending respiratory failure.
- Checking the child's weight.
- Increases glucagon secretion.
- Mimics incretin hormones, stimulating glucose-dependent insulin release, suppressing glucagon, and slowing gastric emptying.
- Increases glucose absorption.
- Decreases insulin sensitivity.
- Risk of hypernatremia.
- Risk of accidental parathyroid gland removal, leading to hypoparathyroidism and life-threatening hypocalcemia.
- Risk of hyperkalemia.
- Risk of hypoglycemia.
- Bowel obstruction.
- Inflammation and autodigestion of the pancreas by activated enzymes.
- Gastric acid reflux.
- Appendicitis.
- Oral medications for blood sugar.
- Initiation of renal replacement therapy (dialysis or kidney transplant) to sustain life and manage complications.
- Only dietary modifications.
- No urgent intervention needed.
- Increased platelet count.
- Impaired synthesis of clotting factors by the damaged liver.
- Increased fibrinolysis.
- Decreased red blood cell production.
- Vasodilation.
- Thrombosis formation on the ruptured plaque, leading to partial or complete coronary artery occlusion.
- Spasm of coronary artery.
- Increased myocardial oxygen supply.
- Increased potassium intake.
- Renal potassium wasting due to increased bicarbonate and volume depletion, and intracellular shift of potassium.
- Decreased aldosterone.
- Decreased renal excretion.
- To immediately start dialysis.
- To identify and urgently relieve the obstruction to urinary outflow (e.g., catheterization, stone removal).
- To administer loop diuretics.
- To prescribe antibiotics.
- To stimulate glucagon release.
- To replace the absolute deficiency of endogenous insulin.
- To decrease insulin resistance.
- To delay glucose absorption.
- Viral infection.
- DNA damage and mutations in lung cells induced by carcinogens in tobacco smoke.
- Autoimmune reaction.
- Bacterial infection.
- Rapid ventricular filling in a dilated ventricle.
- Atrial contraction against a stiff, non-compliant ventricle.
- Valvular regurgitation.
- Pericardial friction rub.
